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A-Lipoic Acid Has the Potential to Normalize Copper Metabolism, Which Is Dysregulated in Alzheimers Disease

Endogenous antioxidant defence system in rat liver following mercury chloride oral intoxication

Pharmacological targeting of endoplasmic reticulum stress in disease

Together, these results suggested a potential cooperativity of BRD4 and ASXL1 that co-occupy transcriptionally active promoters

Whether this mechanism is commonly employed for a variety of substrates and whether monothiol mechanisms (i) and (ii) are really relevant for the reduction of other non-glutathione disulfide substrates remains to be analyzed